NDL is NDL.
NDL is based on nitrogen loading. The more nitrogen in the gas you are breathing the quicker the ongassing. So the significance of nitrox is not the added O2 (mod/oxtox is the limiting factor here) but in the lower percentage of nitrogen.
So if you lower the percentage of nitrogen in your breathing gas you can extend your NDL. But diving to the limit of NDL gives the same risk of DCS, air or nitrox.
Yeah, that's the part I don't quite buy: I'm pretty sure my metabolism won't burn more oxygen just because I breathe more in -- I wish my body worked that way and I could eat more and just metabolize it all away instead of it inflating my spare tire and ldl count. O2 I don't burn should act just like the "inert gasses" traditionally considered by the models. These guys, for instance: ARTICLES | Journal of Applied Physiology gave O2 up to 40% of the risk of N2 up to 1.3 PO2 a decade ago. That's a 60% reduction in the extra 10-15% of O2 in your EAN mix, which is great, but I wonder if it's enough to trump things like PFO or even a bad hangover with hypotermia and some arterial plaque on top..