It seems to me that most ccr accidents involved a lack of o2 rather than too much.
I was taught to bail out straight away when the o2 in the loop is over 1.6. Than flush and see if I can go back on the loop. Would you stay on the loop and try to fix it while your cells show more than 1.6? Unless it's just a 2 second spike or so.
There have been quite a few CCR deaths involving O2 toxicity. How many? I dont know the exact numbers or enough to say if one is more common.
The difference is hypoxia is considered a "silent killer". You pass out and drown. Some will argue you might notice decreased loop volume but I dont buy that argument.
O2 toxity (hyperoxia) may or may not have warn signs associated with Central Nervous System oxygen (CNS) toxicity such as visual changes (especially tunnel vision), tinnitus , nausea, twitching (especially of the face), and dizziness.
You may remember hearing VENTID which is used to remember the symptoms of Oxygen Toxicity. Visual disturbances, Ear ringing, Nausea, Twitching, Irritability, and Dizziness.
In both scenarios without a loop or regulator in your mouth you pass out and drown. The result is usually the same, death. Both could be survivable provided you don't drown. e.g., gag strap on loop, regulator in your mouth.
Both circle back back to the rebreather matra of "always know your ppO2." Monitoring a handset and HUD to alert you to either impending hypoxia or hyperoxia.
It is my understanding that the most recent death in Roaring River, MI was likely the result of an O2 tox / seizure. Not because of any particular equipment failure but because of a diluent that had too high ppO2 / gas density / narcotic effect for the depth involved.
This is probably a whole other discussion and I'm sure people will disagree with me but I won't immediately bailout at ~1.6 ppO2 but it's certainly going to get my undivided and immediate attention.
Hyperoxia caused by breathing oxygen at elevated partial pressures usually has a bunch of different variables, including actual ppO2, time and length of dosage, workload, gas density, individual tolerances to oxygen, etc.
For example: NOAA's maximum CNS recommendation is exposure of 1.6 ppO2 for 45 minutes. We probably all know people who have ended dives way over 100% CNS but I am just using that as an example.
Being at ppO2 1.6 (or even 1.7) is not an instant death sentence but it should damn well get your full attention. If I looked down at my handset and suddenly saw my ppO2 at 2.0 then I'd probably immediately bailout and go into troubleshooting mode. My brain would have some questions. How long have I been at 2.0? Why didnt I notice it sooner? What's my loop / counterlung volume? What's my pressure in my 3L bottle of O2?