I'm going above my pay grade here but I'll give this a shot. Others more medically inclined, please feel free to weigh in. I may also try to get Karl Huggins from the Catalina Chamber to respond but he's manning a polling place today so it would be tomorrow at the earliest.
My initial disclaimer is that I think I'm phrasing all this correectly but I won't promise it 100% spot-on.
Do you have any research that suggests more emboli are created with aveoli bursting compared to ascending and emboli created solely by the bubbles already in your bloodstream expanding?
By definition, an embolus in your bloodtream released from the your lungs (alveoli) is pretty big.
HUGE in comparison to nitrogen bubbles. And the reasons these can be so dangerous is that they're big enough to stop blood flow all together.
Bends is different. Excess nitrogen absorbed by the body diffuses back into the bloodtsream, is carried to the lungs, diffuses back into a gaseous state, and is exhaled. When it's coming out of solution faster than the process can handle (bends) then nirtrogen bubbles form in the bloodtsream. But they're
little, teeny-tiny bubbles. (Think of the difference between a basketball - embolus - and a BB - bends.) These bends bubbles are small enough to travel freely through the body but they get lodged in the tiny, lttile, narrow capillaries which is what causes the joint pain. If they lodge against nerves, that's what causes neurological deficit.
I'm skeptical about this. Sorry. Thank you for explaining it better though.
No problem.
As we know there are bubbles in our body from a recreational dive. To say you can go as fast as you like as long as your lungs dont go above 100% you wont have an embolism is something I don't agree with (or well depending on time depth deco obligation etc). It would be like looking at only the lung injury component.
Yes, I thought I made it clear I was
ONLY talking about the AGE/lung-injury component. (I'm not advoctaing you ascend really fast. I'm only saying that, strictly from an AGE perspective, you can do so without embolising.) I think I even pointed out in the post that because ascent rate is part of decompression, a rapid ascent can cause a bends hit in an otherwise OK profile.
Bends and embolsim are two very different things. I think you're either combining them into one or confusing the two. They're apples and oranges. In it's simplest form, embolism is a result of a breath-holding ascent while on scuba. Bends is result of depth & time on scuba, absorbing more nitrogen than your body can safely outgas and coming back to surface pressure.
Most emboli don't cause immediate death....
Where did you hear that from??? Although you're technically correct . . . You don't embolise and immediately die . . . embolisms manifest themsevles relatively quickly (usually within minutes of surfacing) and are
ALWAYS consdered life-threatening. They require on-site emergency first-aid oxygen, frequently CPR and life support, and immediate treatment in a qulaified hyperbaric chmaber.
While there are instances of bends going untreated and eventually resolving with no apparent after-effects (skin bends is an example that may not require chamber treatment), I don't know of any verified embolsim case where the person didn't get treated and survived. Conversely, there are many cases of embolism where the diver died, whether they got treatment or not.
- Ken