I have a bit of a theory that CNS deression from N2 reduces the likeihood of convulsions on air at high ppO2 compared to the same ppO2 on trimix without the CNS depression. Its not really provable due to the vagueries of convulsions in the first place. But does potentially explain the capacity to do pretty substantial air dives with sleepiness being the overriding symptom instead of excitability, twitching or convulsion type stuff.
Deep air probably makes O2 toxicity more likely d/t other risk factors: cold, and especially elevated blood pCO2 levels. The record-setters have a pretty high fatality rate - I think it's reasonable to assume that O2 toxicity is a factor.