Dr Deco
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Hello Artimas:
Limits
Flying after diving should really be approached conservatively by most divers. Duke University showed that short surface intervals of only a few hours produced what might be termed random occurrences of DCS without a clear pattern. In my experience, this is what is found when one approaches a DCS limit/range, where the free-gas phase in the body is beginning to grow sufficiently large to cause obvious problems. [Before then, the gas phase was not large enough to result in clinical symptoms.]
The NDLs in tables are at much reduced loads and DCS does not appear under normal conditions.
Reduced Pressure DCS
What we are finding in reduced pressure [altitude] cases is that the body has large [a few microns] tissue microbubbles already present it is not a matter of forming new bubbles [as, for example, in the metastable limit]. These microbubbles have come from gas loads and musculoskeletal activity prior to altitude depress resulting in hydrodynamic cavitation. This is a random [mathematically termed as chaotic] process and cannot be statistically treated unless one is a very low gas loads and/or no microbubbles present.
The number of these bubbles formed during activity can be different for each diver. Some people may be very active, hauling suitcases and gear, sometimes even shortly after surfacing when the propensity to bubble formation is greatest. Surface activity was not included in the Duke University study.
This is handled by advising divers to allow a sufficiently long duration between diving and flying. I would definitely not cut short the surface interval in the dive and fly situation.
Rapid Depressurization
Unfortunately, in airplanes, rapid depressurization is a problem that could though rarely occur. As I wrote a few weeks ago, depressurization at high altitude should not result in DCS problems if the plane descends. This conclusion is based on (historical) statistics from NASA laboratory studies where slow depressurization is the norm. Rapid depress would not be performed on human test subjects because of the possible great risk.
A recent paper [1] indicates that repaid depress can result in DCS that is more traceable to barotrauma and gas embolism than tissue bubble formation/growth. Note that if these individuals also had a sizable tissue gas load, their outcome might have been more severe as the emboli could grow and interfere considerably with local circulation.
Direct formation of gas bubbles in the arterial circulation with rapid depressurization did not occur in studies with sheep that I performed several decades ago [2]. The chamber was modified to allow assents of 10 ft/sec. We can assume therefore that the aircrew experienced barotrauma.
I would definitely not cut short the surface interval in the dive and fly situation and not ascend to altitude any more than necessary.
Dr Deco :doctor:
References :book:
[1] Johnston MJ. Loss of cabin pressure in a military transport: a mass casualty with decompression illnesses. Aviat Space Environ Med 2008; 79:429-32.
Presented here is the sudden cabin depressurization of a military C-130 aircraft carrying 66 personnel. They suffered a depressurization from 700 ft (2,134 m) to 24,000 ft (7,000 m), resulting in a potential 66-person mass casualty. The aircrew were able to descend to below 3049 m in less than 5 min. They landed in the Kingdom of Bahrainthe nearest hyperbaric recompression facility. Three cases of peripheral neurologic DCS and one case of spinal DCS were identified. Limited manning, unique host nation concerns, and limited available assets led to difficulties in triage, patient transport, and asset allocation. These led to difficult decisions regarding when and for whom to initiate ground level oxygen or hyperbaric recompression therapy.
[2] MR Powell, MP Spencer, MT Smith. In situ arterial bubble formation and atraumatic air embolism. Undersea Biomed. Res. 9, (1), Suppl., 10 (1982).
Limits
Flying after diving should really be approached conservatively by most divers. Duke University showed that short surface intervals of only a few hours produced what might be termed random occurrences of DCS without a clear pattern. In my experience, this is what is found when one approaches a DCS limit/range, where the free-gas phase in the body is beginning to grow sufficiently large to cause obvious problems. [Before then, the gas phase was not large enough to result in clinical symptoms.]
The NDLs in tables are at much reduced loads and DCS does not appear under normal conditions.
Reduced Pressure DCS
What we are finding in reduced pressure [altitude] cases is that the body has large [a few microns] tissue microbubbles already present it is not a matter of forming new bubbles [as, for example, in the metastable limit]. These microbubbles have come from gas loads and musculoskeletal activity prior to altitude depress resulting in hydrodynamic cavitation. This is a random [mathematically termed as chaotic] process and cannot be statistically treated unless one is a very low gas loads and/or no microbubbles present.
The number of these bubbles formed during activity can be different for each diver. Some people may be very active, hauling suitcases and gear, sometimes even shortly after surfacing when the propensity to bubble formation is greatest. Surface activity was not included in the Duke University study.
This is handled by advising divers to allow a sufficiently long duration between diving and flying. I would definitely not cut short the surface interval in the dive and fly situation.
Rapid Depressurization
Unfortunately, in airplanes, rapid depressurization is a problem that could though rarely occur. As I wrote a few weeks ago, depressurization at high altitude should not result in DCS problems if the plane descends. This conclusion is based on (historical) statistics from NASA laboratory studies where slow depressurization is the norm. Rapid depress would not be performed on human test subjects because of the possible great risk.
A recent paper [1] indicates that repaid depress can result in DCS that is more traceable to barotrauma and gas embolism than tissue bubble formation/growth. Note that if these individuals also had a sizable tissue gas load, their outcome might have been more severe as the emboli could grow and interfere considerably with local circulation.
Direct formation of gas bubbles in the arterial circulation with rapid depressurization did not occur in studies with sheep that I performed several decades ago [2]. The chamber was modified to allow assents of 10 ft/sec. We can assume therefore that the aircrew experienced barotrauma.
I would definitely not cut short the surface interval in the dive and fly situation and not ascend to altitude any more than necessary.
Dr Deco :doctor:
References :book:
[1] Johnston MJ. Loss of cabin pressure in a military transport: a mass casualty with decompression illnesses. Aviat Space Environ Med 2008; 79:429-32.
Presented here is the sudden cabin depressurization of a military C-130 aircraft carrying 66 personnel. They suffered a depressurization from 700 ft (2,134 m) to 24,000 ft (7,000 m), resulting in a potential 66-person mass casualty. The aircrew were able to descend to below 3049 m in less than 5 min. They landed in the Kingdom of Bahrainthe nearest hyperbaric recompression facility. Three cases of peripheral neurologic DCS and one case of spinal DCS were identified. Limited manning, unique host nation concerns, and limited available assets led to difficulties in triage, patient transport, and asset allocation. These led to difficult decisions regarding when and for whom to initiate ground level oxygen or hyperbaric recompression therapy.
[2] MR Powell, MP Spencer, MT Smith. In situ arterial bubble formation and atraumatic air embolism. Undersea Biomed. Res. 9, (1), Suppl., 10 (1982).