Foxfish
Contributor
Thanks Michael. That gives a bit of insight into where the defence was heading.
I found a couple of things interesting. Firstly the time taken to die from asphyxiation is greater when underwater. When doing a freediving course I was taught about the mammalian dive reflex which we humans posess. It is triggered by the presence of water on the face and water pressure and causes the hearbeat to slow and hence reduces oxygen consumption. Extreme cold can also extend the period until a person becomes clinically dead.
Edmonds comments appear to be in brief note form and require some deciphering.
This comment suggests that the time to drown after aspiration of saltwater is less than 1 minute. I'd be interested to know what the 1 minute refers to and whether this time is greater than a typical drowning. This may be due to the laryngospasm occurring in normal drowning but not in saltwater aspiration.
I found an article on saltwater aspiration here:
Saltwater Aspiration Syndrome
It states that Dr Edmonds first described the condition. It goes on to claim however that it would be unlikely to be a cause of death.
Quote from the article:
In saltwater drownings and saltwater "wet" near-drownings (those that involve aspiration), the hypertonicity of the aspirated fluid draws intravascular fluid into the already fluid-filled alveoli, resulting in ventilation-perfusion abnormalities and intrapulmonary shunting. Intravascular hypovolemia, hemoconcentration, and electrolyte abnormalities can result, although this is not usually seen clinically in near-drowning survivors because they rarely aspirate enough water to produce these effects. It is doubtful that there is enough volume aspirated through a regulator to cause significant electrolyte abnormalities.
Salt water aspiration sounds a lot like pulmonary aspiration. In the following link it states that death from pulmonary aspiration can occur within a couple of minutes.
Pulmonary aspiration - Wikipedia, the free encyclopedia
Consequences of pulmonary aspiration range from no injury at all, to chemical pneumonitis or pneumonia, to death within minutes from asphyxiation. These consequences depend in part on the volume, chemical composition, particle size, presence or absence of infectious agents, and underlying health status of the person.
After looking at these references I re-read the comments in McFadyen's post from Edmonds on the condition. They are posted below.
My interpretation of this is that salt water aspiration is when you inhale air from your regulator with an amount of water that forms tiny droplets in the air stream. These small droplets get inhaled into the tiny sacs in you lungs called alveoli (I'll call them sacs) where the exchange of oxygen and carbon dioxide take place. The salt water droplets reduce the effectiveness of the oxygen/carbon dioxide exchange in the sacs. (I don't understand how!). This results in a reduction of oxygen transferred to the blood stream. If your oxygen level falls to dangerously low levels you are said to be hypoxic. High levels of carbon dioxide makes you want to breathe. The reduction in oxygen levels and increase in carbon dioxide levels makes you want to breath more. The increased breathing rate doesn't really help you get the oxygen where it is needed and the increased breathing rate increases the amount of water spray that is inhaled into the lungs. If this continues for long enough the victim will become unconscious, have 'samba' convulsions and then die. Salt water in the lungs also aggravates the sacs. A delayed affect of inhaling salt water is fluid from blood in the sac tending to collect in the sacs. This fluid plus mucous plus the seawater in the lungs gives the appearance that the person drowned when in fact they could have died from saltwater aspiration. Edmond's believes saltwater aspiration occurs in 37 % of fatalities in recreational divers.
That said, I thought the person who did the autopsy was adament that Tina drowned in the recent testimony given in court.
(Edit: Added comments about saltwater aspiration from Edmonds)
I found a couple of things interesting. Firstly the time taken to die from asphyxiation is greater when underwater. When doing a freediving course I was taught about the mammalian dive reflex which we humans posess. It is triggered by the presence of water on the face and water pressure and causes the hearbeat to slow and hence reduces oxygen consumption. Extreme cold can also extend the period until a person becomes clinically dead.
Edmonds comments appear to be in brief note form and require some deciphering.
Her problem was from aspiration leading to hypoxia, leading to unconsciousness. PROB < 1 minute"
This comment suggests that the time to drown after aspiration of saltwater is less than 1 minute. I'd be interested to know what the 1 minute refers to and whether this time is greater than a typical drowning. This may be due to the laryngospasm occurring in normal drowning but not in saltwater aspiration.
I found an article on saltwater aspiration here:
Saltwater Aspiration Syndrome
It states that Dr Edmonds first described the condition. It goes on to claim however that it would be unlikely to be a cause of death.
Quote from the article:
In saltwater drownings and saltwater "wet" near-drownings (those that involve aspiration), the hypertonicity of the aspirated fluid draws intravascular fluid into the already fluid-filled alveoli, resulting in ventilation-perfusion abnormalities and intrapulmonary shunting. Intravascular hypovolemia, hemoconcentration, and electrolyte abnormalities can result, although this is not usually seen clinically in near-drowning survivors because they rarely aspirate enough water to produce these effects. It is doubtful that there is enough volume aspirated through a regulator to cause significant electrolyte abnormalities.
Salt water aspiration sounds a lot like pulmonary aspiration. In the following link it states that death from pulmonary aspiration can occur within a couple of minutes.
Pulmonary aspiration - Wikipedia, the free encyclopedia
Consequences of pulmonary aspiration range from no injury at all, to chemical pneumonitis or pneumonia, to death within minutes from asphyxiation. These consequences depend in part on the volume, chemical composition, particle size, presence or absence of infectious agents, and underlying health status of the person.
After looking at these references I re-read the comments in McFadyen's post from Edmonds on the condition. They are posted below.
The collection of sea water in the regulator may produce a bubbling or “wet” sensation during inspiration, and a nebulised spray is inhaled into the lungs. This has two major consequences. Shunting of the pulmonary circulation across poorly aerated alveoli rapidly results in hypoxia. Any concomitant reduction of carbon dioxide exhalation is overcome by the body increasing the minute ventilation (a natural physiological response to carbon dioxide) – thereby aggravating the over-breathing but not appreciably correcting the hypoxia. The hypoxia induces fatigue, with unconsciousness, then hypoxic spasms and death in severe cases, The cause of death is usually attributed, at autopsy, to drowning. The lungs become fluid filled and heavy with sea water, mucus and oedema.
A somewhat more delayed effect is the accumulation of osmotic fluid in the lungs, from the blood, aggravating the pulmonary oedema, and the other clinical effects, associated with drowning.
Salt water aspiration is a prelude to 37% of the recreational diving fatalities. In some series it is incorporated, incorrectly, as "asphyxia"."
My interpretation of this is that salt water aspiration is when you inhale air from your regulator with an amount of water that forms tiny droplets in the air stream. These small droplets get inhaled into the tiny sacs in you lungs called alveoli (I'll call them sacs) where the exchange of oxygen and carbon dioxide take place. The salt water droplets reduce the effectiveness of the oxygen/carbon dioxide exchange in the sacs. (I don't understand how!). This results in a reduction of oxygen transferred to the blood stream. If your oxygen level falls to dangerously low levels you are said to be hypoxic. High levels of carbon dioxide makes you want to breathe. The reduction in oxygen levels and increase in carbon dioxide levels makes you want to breath more. The increased breathing rate doesn't really help you get the oxygen where it is needed and the increased breathing rate increases the amount of water spray that is inhaled into the lungs. If this continues for long enough the victim will become unconscious, have 'samba' convulsions and then die. Salt water in the lungs also aggravates the sacs. A delayed affect of inhaling salt water is fluid from blood in the sac tending to collect in the sacs. This fluid plus mucous plus the seawater in the lungs gives the appearance that the person drowned when in fact they could have died from saltwater aspiration. Edmond's believes saltwater aspiration occurs in 37 % of fatalities in recreational divers.
That said, I thought the person who did the autopsy was adament that Tina drowned in the recent testimony given in court.
(Edit: Added comments about saltwater aspiration from Edmonds)
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