Mr Carcharodon
Contributor
Have there been studies that define the detailed mechanisms that cause DCS? It seems that there have been a number of threads lately about symptoms showing up some time after dives. The posters have been concerned that symptoms they have may be DCS. It seems many people do not have a clear understanding of the underlying mechanisms. But then does anyone have a clear understanding?
I have assumed that the mechanisms are hypoxia which results from reduced perfusion due to the presence of bubbles. Also I assume there is the potential for trauma due to intracellular and extracellular bubble formation. But are bubbles the whole story? What has been done experimentally to validate the mechanisms?
I have assumed that the mechanisms are hypoxia which results from reduced perfusion due to the presence of bubbles. Also I assume there is the potential for trauma due to intracellular and extracellular bubble formation. But are bubbles the whole story? What has been done experimentally to validate the mechanisms?