Diver-Drex
Contributor
Ok, Table 6 will do it. But we can agree that those exposures are well outside normal recreational diving?
So the point being discussed in this thread is that breathing EAN to prevent DCS during recreational dives doesn't make sense because of the risk of pulmonary toxity. The fact that the treatment for DCS is O2 at over twice the accepted max ppO2 is hardly helping that argument. I would say that it clearly indicates exactly the opposite. O2 at concentrations above 21% as a way to decrease the nitrogen level in the breathing gas is an excellent counter to the affects of inert gas loading, both prophylacticaly and as treatment. And that it is so effective that the risks associated with it, CNS and pulmonary toxicity, are readily accepted. It has its limitations and cautions, but it works.
I've known this to be the case for 30 years, I'm not sure what has changed. I'm open to new information to bring into question EAN use, it just hasn't been brought up here.
So the point being discussed in this thread is that breathing EAN to prevent DCS during recreational dives doesn't make sense because of the risk of pulmonary toxity. The fact that the treatment for DCS is O2 at over twice the accepted max ppO2 is hardly helping that argument. I would say that it clearly indicates exactly the opposite. O2 at concentrations above 21% as a way to decrease the nitrogen level in the breathing gas is an excellent counter to the affects of inert gas loading, both prophylacticaly and as treatment. And that it is so effective that the risks associated with it, CNS and pulmonary toxicity, are readily accepted. It has its limitations and cautions, but it works.
I've known this to be the case for 30 years, I'm not sure what has changed. I'm open to new information to bring into question EAN use, it just hasn't been brought up here.
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