What are your qualifications in hyperbaric medicine?
The reason I'm asking is that I like to know if the person formulating a hypothesis does so based on proper science, or if they're just pulling the hypothesis out of their behind. If a qualified hyperbaric medicine scientist says that something is a reasonable hypothesis, I might well trust them. If an unqualified person does the same, I'll remember the saying about caveat emptor.
To answer your question, I don't have a degree in hyperbaric medicine - but, on the other hand, I don't think what I'm saying is in conflict with anything being presented by those that do?
As a matter of fact, I'm looking at the science we're most often discussing (Nedu, Spisni), and think it could be an interesting bynote to the question.
I'm not saying that some divers need GF0/0 and others GF100/100 - if the "optimal" balance is, say, 50/50, bene - and if there is a difference it may be as little of a difference as +/- 1 point. Or less. Or more. Or nothing at all.
Do
you feel it's a radical idea that one diver might reach "optimal" results from stopping first at one given depth, and another diver at another depth (based on their CCL5-response)?
In either case, it's only a sidenote (and hypothetical), and doesn't really impact the main course of the conversation. I simply thought it'd be an interesting idea to play with as a question was raised if there's any possibility that GI3/AG might have physiological attributes that mean they'd actually feel better from stopping (somewhat) deeper, compared to another tentative sample population;
I want to be 100% clear here, that I don't hold that to take anything away from the NEDU-study, the Spisni-study or any other study, nor reduce their relevance.
Quite the opposite, in fact.
In either case, while it's a layman's appraisal that the idea isn't radical, it seems to be shared by experts in the field (pardon the lack of proper referencing below, but it's from the
Spisni-study report);
"Increased circulating chemokines and higher bubble grades may be two phenomena that are physiologically disconnected. That is, bubble development and the increased inflammation likely induced by vascular modifications might be independent phenomena, both able to enhance divers' susceptibility to develop DCS. However, endothelial physiology which also depends on individual genetics, is certainly linked to the inflammatory response trigger elicited by circulating bubbles. Further studies will be necessary to correlate circulating chemokines with the differences in accepted measures of decompression stress such as the increase of DCS, VGE evolution or different dive profiles with unequivocal differences in decompression stress."
Again, I don't think what I'm saying means anything to the value of neither NEDU or Spisni, and I
don't mean for this to come off as a counterargument in the debate - more of a bynote, rather.
.
