Hello Antonio,
I think that there is no theory universally accepted by the scientific community and scientifically validated by a serious test protocol with a significant number of samples.
I don't agree. The relevant theories on ICD that are "accepted by the scientific community" are reasonably clearly articulated in the Lambertson and Idicula paper you cited. Those theories have, however, been misinterpreted by the technical diving community. There has also been one "serious test protocol with a significant number of samples" in a recent study by NEDU in which they conducted 100 dives with what you would consider to be extremely provocative heliox to air switches and demonstrated that even the form of DCS (inner ear) we link with gas switches is at least relatively uncommon (no cases in 100 dives with radical gas switches). You can read that study
here.
If I'm not wrong, there was test made by Duke university in Washington DC, that shown that divers breathing Heliox at 7 ata had problems immediately after gas switches.
The same thing was experienced by Comex divers in Hydra program
You need to be much more precise about this before presenting it as evidence that what you say is correct. For example:
What gas switches, and what environment were the divers in? I have already pointed out that ICD can occur in environments relatively unique to certain saturation scenarios (such as helium to nitrogen switches when the diver occupies a helium filled environment). But these are not relevant to what technical divers do (unless, for example, you put helium in your drysuit).
What "problems"?. I have pointed out that inner ear DCS can occur after helium to nitrogen switches and may be partly due to augmented supersaturation of the inner ear due to counter diffusion occurring after the switch. Inner ear DCS would therefore not be unexpected, but your narrative essentially implies that helium to nitrogen switches may provoke bubble formation in a wide range of tissues.
They noticed that changing gas mix during decompression caused unexpected bubble formation and it was necessary to recompress them.
They changed decompression protocol, making gas changes more progressive and they got correct decompression.
I would suggest that you find the references, read them, and quote them very precisely if you are going to use them to support your position.
So, So I'm aware that there is not a widely accepted theory that can exactly describe and modelize ICD issues
Again, I do not agree with that. David Doolette and I did exactly that for the one form of DCS thought associated with helium to nitrogen switches in bounce diving decompressions. [1] Importantly, the explanation for the vulnerability of the inner ear in this setting is not related to the mechanisms you describe in your article.
That said, what is your position about ICD risks prevention? Do not worry at all about the limitation of N2 raise to He fall? Do you think this is useless or dangerous? If yes, why?
In my personal opinion this can reduce ICD risks and in any case does not add any additional risk. So why do not apply this protocol?
These are valid points. Avoiding gas switches or making them less dramatic may reduce the risk of inner ear DCS. Ross and I have previously argued about what tissues are affected by ICD, but I have not had a fundamental objection to his inclusion of an ICD warning in his deco software because it is probably harmless, and may reduce inner ear DCS risk; possibly not by much in view of the Doolette NEDU data cited above, but maybe it helps. The same would be true of your approach. My main objection to your article is that it presents a detailed mechanistic view of ICD that is both unreferenced to supporting data and inaccurate.
Simon M
1. Doolette DJ, Mitchell SJ. Biophysical basis for inner ear decompression sickness. J Appl Physiol 2003;94(6):2145-50