Claudia, I suspect that with the diffusion difficulties presented in pulmonary edema, an inspired ppO2 of 1.6 or even 2.0 would not translate into an arterial pO2 anywhere near that high. As I said, in all accounts I have read of significant cases of this illness, the patients have been cyanotic at the surface, which represents a major desaturation of hemoglobin.
It wasn't a recommendation, but it was a thought. I suspect that, if I felt I had IPE and I was terribly short of breath and facing a deco obligation, I'd switch to my hottest mix as soon as possible and see if it alleviated the symptoms at all. As you know, air hunger is usually CO2, and by the time you are short of breath from hypoxia, you are hypoxic indeed. Of course, the dyspnea of IPE may also be in part increased work of breathing with stiff, fluid-filled lungs, and in severe cases, there may be enough interference with gas transport to raise the CO2 as well.