Scuba-Stu
Contributor
I do not know much about decompression theory but I have a couple questions about some things Ive have read lately so excuse me if these are dumb questions. First, Some people on this board and elsewhere have stated that some degree of "bubble formation" is ok or at least tolerable. For example, I just read the following by Eric Maiken,
"Any diver who has been routinely monitored with a Doppler meter can tell of times they bubbled--even after following a conservative ascent schedule. As unnerving as the experience is, the first thing that you wonder after hearing low-grade bubbles is "why don't I feel bent?" The answer may be that the body has the ability to handle small amounts of bubbles without undue stress. There is substantial evidence that the body's tissues contain cavities and bubble nuclei before making a dive. These preexisting voids are activated into growth with the application and reduction of pressure associated with diving. Bubbles are especially prone to growth when surrounding tissue is heavily loaded with dissolved gas, such as might be expected following long, deep or repetitive dives." (Maiken, 1995)
Does any one know what keeps these "bubbles" in the tissues? Couldn't even a microscopic bubble in the blood stream could cause some kind of stroke?
Maiken, Eric. (1995). BUBBLE DECOMPRESSION STRATEGIES. Bubble Decompression Strategies
My next question has to do with PFO testing. Has any one read the following artical by Moon & Bove http://rubicon-foundation.org/dspace/bitstream/123456789/4018/1/15568413.pdf ?
What do you think about it?
"Any diver who has been routinely monitored with a Doppler meter can tell of times they bubbled--even after following a conservative ascent schedule. As unnerving as the experience is, the first thing that you wonder after hearing low-grade bubbles is "why don't I feel bent?" The answer may be that the body has the ability to handle small amounts of bubbles without undue stress. There is substantial evidence that the body's tissues contain cavities and bubble nuclei before making a dive. These preexisting voids are activated into growth with the application and reduction of pressure associated with diving. Bubbles are especially prone to growth when surrounding tissue is heavily loaded with dissolved gas, such as might be expected following long, deep or repetitive dives." (Maiken, 1995)
Does any one know what keeps these "bubbles" in the tissues? Couldn't even a microscopic bubble in the blood stream could cause some kind of stroke?
Maiken, Eric. (1995). BUBBLE DECOMPRESSION STRATEGIES. Bubble Decompression Strategies
My next question has to do with PFO testing. Has any one read the following artical by Moon & Bove http://rubicon-foundation.org/dspace/bitstream/123456789/4018/1/15568413.pdf ?
What do you think about it?