Not at all. But there is still no requirement to call I vs II; you'd describe it as DCS (or DCI) with subjective neuro findings alone said dermatome, or with decreased sensation/strength, whatever if there are objective findings. Even if you come up from a dive paralyzed from the waist down, crystal clear traditional II, these days odds are it'd be "severe neurologic DCS with *describe loss of motor/sensory* at the x spinal level, bladder retention, blah blah."
Now, in a very clear example sure, you can say II. But it doesn't really add anything to the diagnosis, it doesn't guide treatment, etc. The description is clearer than calling a type, and you treat based on symptoms and response to treatment, not what type you called it. And with the majority of bends being not quite so cut&dry about what system is/isn't affected, the divide between traditional I and II gets mucked up and becomes completely useless.
And I agree with scorpio, sometimes you just get bent. Bent a guy on a certain profile recently, in the chamber - he's done this same profile before multiple times, multiple other divers have done it. Chamber so there was no difference in rates of ascent, no bouncing up and down; but one dive, boom, dude gets a hit.
Luck of the draw, sometimes; nobody understands how the hyperbaric environment affects our bodies exactly, at the immune and genetic level. We know it activates a whole bunch or transcription factors, but what do they all do? And if you're not 100% at the time of your dive for whatever reason, how does that affect you - we have associations for what increases risk, like dehydration, but not the exact mechanism for why.
