Here is the link to the whole article.
It is a complex article citing many sources. It also presents a complex situation. Take, for example, this quote:
Dumić et al. [147] reported that regular recreational SCUBA diving promotes an anti-inflammatory status, thus contributing to cardioprotection and conferring multiple health benefits. Divers who performed five dives, one per week, at a depth of 20–30 m that lasted 30 min, manifested IgG and TPP N-glycosylation alterations toward anti-inflammatory status over the whole study period with an increase in monogalyctosylated and core-fucosylated IgG N-glycans and a decrease in galactosylated TPP N-glycans. On the other hand, military combat swimmers (O2 divers) are regularly exposed to hyperbaric hyperoxia in addition to intensive endurance training intervals and, therefore, are exposed to extreme levels of oxidative stress. Compared to controls, they exhibited a proinflammatory immune status exemplified by an elevated number of CD4+CD25+ T cells, the elevated expression of proinflammatory cytokine IL-12, and the diminished expression of anti-inflammatory TGF-β1 [148]. Supported by decreased basal gene expression and the prolonged upregulation of anti-oxidative HO-1, these data suggest that higher oxidative stress levels, as those present under intermitted hyperbaric hyperoxia conditions, e.g., through oxygen diving, promote a higher inflammatory immune status.
The paper's focus on problems with high oxygen pressures is dealing with very long exposures to very high PPO2s, something that has been well understood for many decades. They reference prolonged exposures to PPO2s of 1.0 or above. Technical divers are taught to take air breaks when doing long decompression stops using pure oxygen. People doing decompression therapy in a chamber also take periodic air breaks.
