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Descending increases the partial pressure of the CO thereby causing more to bind to hemoglobin. Using an arbitrary figure for discussion, 10 ppm CO wouldn't hurt you at 1 atmosphere, as you descend - the effect is increased. Their dive to 23 atmospheres would increase the 10 ppm to an effect similar to 230 ppm. It's not that simple, but it's similar - like diving to depths increases Nitrogen loading as an example.How would ascending release bound CO at an accelerating rate? Or at any rate? I don't think the CO-hemoglobin bond in itself is affected by ambient pressure (not taking the effect of O2 pressure into consideration here), it's more of a chemical thing. From a total CO load perspective alone, the shallower the better. Or is it not?
Ok, so after loading hemoglobin at such high levels, then ascending causes the CO to start being released, therefore the effect if magnified. But the nature of CO poisoning is so complex that it can't really be managed - just avoid it at all costs, even if you have to test tanks for recreational & deeper dives or use lab prepared gases for excessively deep dives.
There is also some truth to that, but the bigger risk is what I described above here.My understanding is that the effect of the CO is masked by the higher ppo2 at greater depths and is revealed as the lower ppo2 at shallower depths doesn't provide the therapeutic effect that it did when higher. Do I have this wrong?