Dr Deco
Contributor
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Hello farsidefan:
You should be cautious about quickly returning to diving since it appears from your posts that your neurological DCS did not completely resolve following two hyperbaric oxygen treatments. This indicates that proper neural reconnections have not been made. [This is assuming that there is not excessive injury, and reconnections are possible.]
A DCS incident will not preclude you from further diving. [For scientific diving, DCS with unresolved neurologic sequelae is a different matter – this is serious.]
What Happened?
You indicate that nitrogen gas loads were not excessive. A possibility is that venous bubbles formed, and they were arterialize. This arterialization could have resulted from:
- - Pulmonary overload [too many gas bubbles in the capillaries of the lungs], or
- - A PFO and a temporary pressure reversal of the normal L-R pressure gradient, or
- - “Valsalva-like maneuvers” [pulling, straining, coughing] that led to passage from the venous to arterial side.
All of this can be exacerbated by too much physical activity.
Now What?
If you did not overly load nitrogen in you tissues, or run around excessively topside, or perform Valsalva-like maneuvers then you could have an anatomical problem [PFO or pulmonary vessels]. The PFO could be repaired but it is usually easier to dive more conservatively.
Hydration
This is not usually a big player in the normal recreational diver. It has been investigated, and results show that very dehydrated animal subjects have a higher risk of DCS – and this includes neurological DCS.
You should be cautious about quickly returning to diving since it appears from your posts that your neurological DCS did not completely resolve following two hyperbaric oxygen treatments. This indicates that proper neural reconnections have not been made. [This is assuming that there is not excessive injury, and reconnections are possible.]
A DCS incident will not preclude you from further diving. [For scientific diving, DCS with unresolved neurologic sequelae is a different matter – this is serious.]
What Happened?
You indicate that nitrogen gas loads were not excessive. A possibility is that venous bubbles formed, and they were arterialize. This arterialization could have resulted from:
- - Pulmonary overload [too many gas bubbles in the capillaries of the lungs], or
- - A PFO and a temporary pressure reversal of the normal L-R pressure gradient, or
- - “Valsalva-like maneuvers” [pulling, straining, coughing] that led to passage from the venous to arterial side.
All of this can be exacerbated by too much physical activity.
Now What?
If you did not overly load nitrogen in you tissues, or run around excessively topside, or perform Valsalva-like maneuvers then you could have an anatomical problem [PFO or pulmonary vessels]. The PFO could be repaired but it is usually easier to dive more conservatively.
Hydration
This is not usually a big player in the normal recreational diver. It has been investigated, and results show that very dehydrated animal subjects have a higher risk of DCS – and this includes neurological DCS.
Gempp E, Blatteau JE, Pontier JM, Balestra C, Louge P. Preventive effect of pre-dive hydration on bubble formation in divers. Br J Sports Med. 2009 Mar;43(3):224-8.
Fahlman A, Dromsky DM. Dehydration effects on the risk of severe decompression sickness in a swine model. Aviat Space Environ Med. 2006 Feb;77(2):102-6.
Dr Deco :doctor:Fahlman A, Dromsky DM. Dehydration effects on the risk of severe decompression sickness in a swine model. Aviat Space Environ Med. 2006 Feb;77(2):102-6.