The question remains though how was the pulmonary toxicity qualified and quantified for the purpouses of recompression table 6’s treatment?
I don't think anyone is alive that can answer that from direct personal knowledge. Table 5&6 appeared in the 1970 US Navy Diving Manual. Table 4 was in use for decades before that with even higher pulmonary OxTox exposure.
My guess is the docs and physiologists at
NEDU considered pulmonary OxTox a minor side-effect of treatment, as they do today. Navy divers would be restricted from diving for 30 days after any DCS treatment and would have to pass a diving physical before getting in the water again. No big deal, they would just work as tenders instead of taking their turn in the dive rotation.
I can only remember having pulmonary OxTox symptoms twice and it went away in an hour or two. YMMV. I doubt is is good for you but smoking or vaping is far worse in the big picture.
There was very little discussion of pulmonary OxTox when I was in Navy Second or First Class diving schools in the early 1970s. We definitely didn't calculate OTUs, but diving medical officers probably could have, if they cared.
Keep in mind that there is a huge difference between emergency DCS treatment and routine diving operations. There was no good reason for the Navy to get all that concerned with OTUs until eCCRs were approved. Surface-supplied HeO
2 divers only had 3 gases available — air, bottom pre-mix, and pure O
2. Outside of training, HeO2 diving itself was effectively only used in emergencies — like submarine rescue or critical salvage like a special aircraft.
There is no reason for the US Navy, and most others, to expose divers to the risks that trimix rebreather "technical" divers routinely incur. They just go to
saturation if the depth and bottom time demands it, or use a ROV.