... A couple of notes. First, even though it's a minor point in the article, we need to differentiate between skin marbling and type I skin rash...
My purpose in referencing the article was only the fact that chamber availability is unreliable. My commercial diving experience has led me to avoid decompression dives when a chamber was not onboard or at least available within a few hours.
... Gene participated in some research in the early 90's that was led by Wayne Gerth of the Naval Experimental Diving Unit. If you go to
Rubicon Foundation, click on "research repository", and search for "surface interval oxygen", you'll find several items that will make interesting reading. The short story is that they developed some procedures by which they could shorten surface intervals by using prophylactic O2, and not increase the theoretical risk of DCS. ...
Very interesting, thanks. In the case of Surface Interval Oxygen Repetitive Dive Tables, would calling it prophylactic O2 be incorrect since it was used as part of the decompression procedure?
This concept occurred to me primarily because of experience using a proprietary commercial Sur-D-O
2 table (Surface Decompression using Oxygen). It shortened the decompression penalties of reped dives pretty dramatically. The unconfirmed rumor going around the gas shack was that the extended time on Sur-D-O
2 made us clean when the seal broke (about the same residual Nitrogen as the start of the day) -- reped dives had longer decompression but that was the rumor. We experienced zero hits over about 150 dives... not statistically conclusive but interesting. The idea of turning the approach around a little for a prophylactic measure was not much of a stretch, but certainly justified asking those who are better informed.
... The other side of the coin is this: I think prophylactic post-dive O2 could be detrimental if you've just made a dive that's going to cause you to develop DCS (which you won't necessarily know). ...
Agreed, but isn't the same is true of any normal repetitive dive? The intent is to following RGBM-based schedules with air or Nitrox deep and O
2 at 20' up -- with no credit for prophylactic O2.
Besides limiting boredom, the "normal" possibility of DCS was a primary reason for full face masks and comms. The downside of full face masks is succumbing to nappy time. Surface O
2 would be easier and lower risk, but I am
assuming would be significantly less effective than at 20'. I am personally inclined to accept these risks over the possibility of taking a hit that far from a chamber. I just don't want to use this procedure based on ill-informed intuition.
... I'll say up front that I don't have any experimental data to back this up, but I think I'm pretty well-grounded empirically. If you complete a normal dive and do not develop DCS, 100% surface O2 will speed up elimination of dissolved inert gas. However, let's say for example that you have a PFO. You complete an aggressive dive and, unbeknownst to you, you arterialize a clinically significant volume of inert gas bubbles when you pull yourself up the ladder...
This is for fun. Aggressive decompression dives are for being paid and having a chamber onboard. I encourage anyone reading this to adopt the same attitude!
... You go on your planned post-dive O2 right away, and the O2 masks (or partially masks) the progressing symptoms of DCS by providing a modicum of anti-inflammatory effect and an increased pressure gradient. It's been proven numerous times that a diver with DCS who becomes asymptomatic after surface O2 is at high risk of symptom recurrence once that O2 is discontinued; this would apply, by extension, to the present scenario. You go off your surface O2, put on your gear and jump in for your second dive. Now, not only do you have DCS, you're about to increase the amount of inert gas that's dissolved in your body. ...
Understood and agreed. I suppose it comes down to probabilities and risk analysis. Although this scenario understandably has the potential to exacerbate a hit from the initial dive, wouldn't prophylactic O2 be more likely to prevent developing symptoms in the first place? The assumption is the surface interval is part of the decompression profile, not just time spent in the water.
On the other hand, when DCS symptoms develop after a normal reped, wouldn't the lower residual Nitrogen (from prophylactic O2) be advantageous?
...Your CCR idea would certainly eliminate more inert gas than simply following a decompression profile or using surface O2, and using a FFM and hard-wired comms would increase the safety margin. However, I think that the same priciple applies here, though arguably to a lesser extent. Also, you'd need to be more mindful of your O2 clock, especially if you're using this procedure over multiple days.
Good point. Since we aren't crediting prophylactic O2, we would not take our computers with us on O
2 repeds. Therefore we could not depend on computers to track our O
2 clock.
Thank you very much for your informative and thought-provoking reply