Thanks a lot. It would be nice to know what experts think about your explanation - if it's aligned with current scientific knowledge or not
@Duke Dive Medicine @Dr Simon Mitchell
Hello,
It is difficult to be definitive on this, and much depends on how you define fast and slow tissues, but I don't think Angelo's theory is strictly correct. The danger implied by a PFO arises from the fact that it allows venous bubbles to enter the arterial circulation. The question is 'where do these venous bubbles come from'? They almost certainly form in the capillary beds of supersaturated tissues, and migrate into the venous blood from there. Very fast tissues are quantitively small, don't remain supersaturated long, and probably contribute little to a sustained venous bubble load after surfacing. On the other hand, slower tissues with a large capacity for dissolving nitrogen (such as fat, possibly skin) almost certainly contribute to sustained release of venous bubbles that we see after diving. These tissues are probably more relevant to the danger imposed by a PFO. However, I would stop short of categorically claiming that fast tissues are never relevant.
JonG1:
Yeah I've had a shoulder hit post closure, with relatively aggressive GFs, repetitive diving and a bit of swell
This is not surprising, since the occurrence of musculoskeletal DCS has never been linked to the presence of a PFO. This implies that musculoskeletal pain in DCS is one of those forms of DCS probably caused by bubbles forming within the tissues themselves; not by bubbles entering the arterial blood across a PFO.
@Frogman82 , as many have pointed out it is certainly possible to have a PFO repaired for the purpose of reducing risk in diving. Several posters on this thread have had it done. However different jurisdictions/health funding models have different perspectives on who should pay for a closure in this context.
I am surprised that a diving physician has given you a blanket 'stop diving' opinion in the absence of repair, because conservative 'low bubble' diving is a recognised option for managing the risk of a PFO in diving. That point is clearly codified in the SPUMS / UK Sport Diving Medical Committee guidelines which you can find
here. The DAN / UHMS consensus says the same thing. There is also some published evidence that these strategies work, which you can find
here and
here. The problem with such an approach is that it is hard to achieve in technical diving, although I note at least one poster has articulated a series of steps taken in tech dives that appear to be working for him or her. Available data also suggest that a PFO closure is likely to be a more effective protective strategy. In general, if a diver on an ambitious technical diving trajectory is tested and found to have a large PFO, I would generally recommend having it closed (to accommodate that sort of diving). However, if someone only wants to dive to 15m to poke around a tropical reef the advice might be quite different.
@ginti and
@Ucarkus I think you both get the idea on this debate you are having - you are just on slightly different sides of the fence. For clarity, we (and I'm virtually certain I speak for Doug Ebersole here too) do not currently recommend
routine screening of technical divers for PFO. However, given that dives to progressively deeper depths are not iso-risk even if the the same decompression algorithm is followed to the letter, we also understand that there may be some technical divers performing deep decompression dives who a motivated to be tested, even if they have never suffered DCS. A theme in my long previous post that Ucarkus linked to was that most of us are not obstructive in the face of such requests, but it is beholden on us to ensure that we inform the diver fully about what they are getting into by opting for testing. I won't repeat that long post here, but after hearing the explanation described therein, there are some divers who choose not to test and some who forge ahead. Its a classic shades of grey area of medicine.
Simon M